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17q21.2-q21.32CNV Type: Duplication


Largest CNV size: 5622415 bp

Statistics Box:
Number of Reports: 1



Summary Information

A de novo duplication within this locus was identified in a 2-year-old male patient presenting with global developmental delay and multiple congenital anomalies from a cohort of 573 individuals from North West Spain with developmental delay/intellectual disability (Quintela et al., 2017).

Additional Locus Information

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References

Major Reports

Title
Author, Year
Report Class
CNV Type

No Major Reports

Minor Reports

Title
Author, Year
Report Class
CNV Type
Copy number variation analysis of patients with intellectual disability from North-West Spain.
Duplication

Cases

Cohort ID
Author, Year
Descripton
Cohort Size
Diagnosis
Age
Gender
CNV Size
Deletion
Duplication
Total CNV's
 quintela_17_DD/ID_discovery_cases
 Galician (NW Spain) patients recruited from the Complexo Hospitalario Universitario de Santiago de Compostela and referred to the Fundacion Publica Galega de Medicina Xenomica for genetic study
 573
 All participants had a clinical diagnosis of idiopathic intellectual disability (ID) or global developmental delay (DD) with or without another medical condition [e.g. autism spectrum disorder (ASD), attention deficit hyperactivity disorder (ADHD), epilepsy, dysmorphic features, and/or congenital anomalies]
 Range, 3 months-18 years
 60.38% Male
 5622415
 0
 1
 1

Controls

No Control Data Available

Cases

Cohort ID
Geographical Ancestry
Discovery Method
Platform
Algorithm
Software
Validation Method
 quintela_17_DD/ID_discovery_cases
  North West Spain
 Array SNP
  Affymetrix Cytogenetics Whole-Genome 2M SNP array, Affymetrix CytoScan HD
 
 Affymetrix ChAS v.1.2.2
 None

Controls

No Control Data Available

Cases

Patient ID
Author, Year
Age
Gender
Primary Diagnosis
Clinical Profile
Cognitive Profile
CNV Start
CNV End
CNV Size
Genome Build
Type Method
Validation
  quintela_17_DD/ID_discovery_cases-caseID_580
 2 yrs.
 M
 Developmental delay
 Multiple congenital anomalies
 Global developmental delay
 41785215
 47486515
  5701301
 GRCh38
 Duplication
 No

Controls

No Control Data Available

Cases

Patient ID
Validation Description
Primary Disorder Inheritence
Inheritence
Family Profile
Disease Segregation
Gene Content
Altered Gene Expression
 quintela_17_DD/ID_discovery_cases-caseID_580
 
 
 De novo
 
 Possibly segregated
 FKBP10,KAT2A,HCRT,RNU7-97P,MIR548AT,MIR5010,PTP4A2P1,NAGLU,HSD17B1P1,MLX,PSMC3IP,ATP5MGP7,HMGB3P27,PLEKHH3,MIR6780A,HMGN2P15,RAMP2-AS1,COA3,MIR6781,AOC2,AOC3,AOC4P,RNU6-287P,RPL27,RND2,RPL21P4,TMEM106A,RNU2-1,RNU2-4P,RNU6-1137P,ARL4D,RNU6-470P,MIR2117,RNU6-406P,RNU6-971P,SOST,CFAP97D1,FAM215A,LINC01976,RNU6-131P,ASB16-AS1,ATXN7L3,MIR6782,SHC1P2,RN7SL507P,RNU6-453P,RUNDC3A,SLC25A39,FAM171A2,RPL7L1P5,RN7SL258P,FZD2,SMCO4P1,RN7SL819P,HIGD1B,RN7SL405P,CCDC103,FAM187A,MIR6783,C1QL1,MIR6784,HEXIM1,HEXIM2,SPATA32,RNA5SP443,MIR4315-1,RDM1P1,DND1P1,MAPK8IP1P2,RPS26P8,SPPL2C,MAPT-IT1,STH,KANSL1-AS1,MAPK8IP1P1,RN7SL656P,RDM1P2,RN7SL199P,FAM215B,RPS7P11,LINC01974,RNU6ATAC3P,MIR5089,RPRML,RN7SL270P,RNU7-186P,JUP,NT5C3B,KLHL10,KLHL11,ACLY,CNP,DNAJC7,NKIRAS2,C17orf113,DHX58,HSPB9,RAB5C,KCNH4,GHDC,STAT5A,CAVIN1,HSD17B1,COASY,TUBG1,TUBG2,CCR10,CNTNAP1,RAMP2,VPS25,WNK4,CNTD1,BECN1,PSME3,LINC00671,G6PC,PTGES3L-AARSD1,AARSD1,PTGES3L,IFI35,VAT1,NBR2,NBR1,MIR2117HG,ETV4,LINC02594,MPP3,CD300LG,PPY,PYY,NAGS,TMEM101,G6PC3,HDAC5,C17orf53,TMUB2,UBTF,SLC4A1,GRN,ITGA2B,LINC01180,MEIOC,CCDC43,DBF4B,ADAM11,EFTUD2,GFAP,KIF18B,DCAKD,PLCD3,ACBD4,FMNL1,MAP3K14-AS1,MAP3K14,ARHGAP27,LRRC37A4P,LINC02210,CRHR1,ARL17B,LRRC37A,WNT9B,LRRC37A17P,MYL4,ITGB3,EFCAB13,MRPL45P2,P3H4,ZNF385C,STAT5B,STAT3,ATP6V0A1,RETREG3,EZH1,BRCA1,CCDC200,LINC00910,DHX8,MEOX1,DUSP3,MPP2,ASB16,RUNDC3A-AS1,GPATCH8,GJC1,PLEKHM1,LINC02210-CRHR1,MAPT-AS1,MAPT,NSFP1,LRRC37A2,ARL17A,NSF,WNT3,GOSR2,THCAT158,RUNDC1,LSM12,NMT1,KANSL1,CDC27,TTC25
 

Controls

No Control Data Available
No Animal Model Data Available
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