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7q22.3-q31.1CNV Type: Deletion


Largest CNV size: 2900000 bp

Statistics Box:
Number of Reports: 1



Summary Information

A de novo 7q22.3-q31.3 deletion affecting the KMT2E gene was identified by aCGH in a 7-year-old male patient presenting with developmental delay, intellectual disability, seizures, and multiple structural brain abnormalities (O'Donnell-Luria et al., 2019).

Additional Locus Information

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References

Major Reports

Title
Author, Year
Report Class
CNV Type
Heterozygous Variants in KMT2E Cause a Spectrum of Neurodevelopmental Disorders and Epilepsy.
Deletion

Minor Reports

Title
Author, Year
Report Class
CNV Type

No Minor Reports

Cases

Cohort ID
Author, Year
Descripton
Cohort Size
Diagnosis
Age
Gender
CNV Size
Deletion
Duplication
Total CNV's
 o'donnell-luria_19_ASD/DD/ID/EP_discovery_cases
 Individuals with de novo microdeletions encompassing the KMT2E gene (one individual had been previously reported in Uliana et al., European Journal of Human Genetics 2010)
 4
 All four cases present with developmental delay and intellectual disability; three cases also present with epilepsy, while one case was diagnosed with ASD
 Range,7-22 years
 50% Male
 2900000
 1
 0
 1

Controls

No Control Data Available

Cases

Cohort ID
Geographical Ancestry
Discovery Method
Platform
Algorithm
Software
Validation Method
 o'donnell-luria_19_ASD/DD/ID/EP_discovery_cases
  European
 aCGH
  Agilent 44B
 
 
 RT-qPCR

Controls

No Control Data Available

Cases

Patient ID
Author, Year
Age
Gender
Primary Diagnosis
Clinical Profile
Cognitive Profile
CNV Start
CNV End
CNV Size
Genome Build
Type Method
Validation
  o'donnell-luria_19_ASD/DD/ID/EP_discovery_cases-case32
 7 yrs.
 M
 DD, ID, and epilepsy
 Developmental milestones: developmental delay (walking at 15 months, first words at 36 months). Language and communication evaluation: absence of fluent speech. Motor and musculoskeletal evaluation: tapered fingers, delayed bone age. Behavioral/psychiatric evaluation: frequent crying, irritability. Epilepsy/seizures: seizures. EEG: left temporo-parietal epileptiform activity and slowing. Brain imaging: reduced supratentorial white matter, ventricolomegaly, corpus callosum hypoplasia, right cerebellar hypoplasia, enlargement of the cisterna magna, choroid plexus cysts of the lateral ventricles. Additional medical history: frequent colitis, unilateral cryptorchidism. Dysmorphic features: sparse eyebrows, deep set eyes, impression of hypotelorism, sagging cheeks, prognathism, macroglossia, large ears, prominent columella. Growth parameters: head circumference + 2.9 SD, weight + 2.5 SD, height + 2.4 SD. Note: this case had been previously reported in Uliana et al., European Journal of Human Genetics 2010.
 Mild intellectual disability (IQ 55)
 104865561
 107768412
  2902852
 GRCh38
 Deletion
 Yes

Controls

No Control Data Available

Cases

Patient ID
Validation Description
Primary Disorder Inheritence
Inheritence
Family Profile
Disease Segregation
Gene Content
Altered Gene Expression
 o'donnell-luria_19_ASD/DD/ID/EP_discovery_cases-case32
 RT-qPCR
 
 De novo
 
 
 RN7SL8P,KMT2E-AS1,RWDD4P1,RNU6-1322P,YBX1P2,DCAF13P1,RNU6-392P,LARP1BP2,RNA5SP236,GPR22,WBP1LP2,LHFPL3-AS2,KMT2E,RINT1,EFCAB10,SYPL1,NAMPT,CCDC71L,PIK3CG,HBP1,DUS4L,BANF1P5,SLC26A4-AS1,SLC26A4,CBLL1,SLC26A3,LINC01004,SRPK2,PUS7,ATXN7L1,LINC02577,PRKAR2B,COG5,BCAP29,LHFPL3,CDHR3
 

Controls

No Control Data Available
No Animal Model Data Available
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